Medico Guides 4th Block (Module No 07) Physiology Guidelines

Prepared by:

                   Alisha Athar (G13)

Compiled by:

                   Hafiz Muhammad Umair Noor (G12)

  • Guyton and Hall Textbook of Medical Physiology 14th Edition (Chapter numbers are mentioned according to this edition)

THE BODY FLUIDS AND KIDNEYS

  • Body fluid compartments
  • ECF comaprtment
  • Iso,hyper,hypo osmotic fluids
  • Hypo and hypernatremia
  • Table 25.4
  • Fig 25.7
  • Edema (intracellular and extracellular edema)
  • Summary of safety factor that prevents edema
  • Functions of kidney
  • Physiological anatomy of kidney
  • Blood supply of kidney
  • Difference between cortical and juxta medullary neprhons
  • Micturition
  • Innervation of bladder(v.imp)
  • Vesicoureteral reflux
  • Ureterorenal reflex
  • Cystometrogram with fig 26.8
  • Micturition reflex (vvvv imp)
  • Facilitation and inhibition of micturition
  • Abnormalities of micturition (vvvvvvv imp)
  • Urinary excretion rate equation
  • Why large amount of solutes are filtered and reabsorbed by kidney
  • Glomerular filtration rate
  • Composition of glomerular filtrate (read)
  • Filtration fraction (definition and equation)
  • Glomerular capillary membrane
  • Why albumin restricted from filtration (imp)
  • Minimal change nephropathy ( proff seq)
  • Determinants of GFR ( proff seq)
  • Fig 27.4
  • Increase glomerular capillary~ filtration coefficient
  • Increase GFR with increase hydrostatic pressure
  • Fig 27.7
  • Table 27.2
  • Renal blood flow(read)
  • Table 27.4
  • Control Of Glomerular filteration and blood flow(compl)
  • Autotegulation of GFR(compl)
  • Tubuloglomerular  feedback
  • Fig 27.11(SEQ)
  • Fig 27.10
  • Blockade of angiotensin formation .. (blue box pg 340)
  • Myogenic autoregulation
  • Fig 27.12
  • Table 27.5
  • Urinary excretion and filtration formula
  • Transcellular pathway and para cellular pathway (active transport on 2nd page of this chapter)
  • Fig 28.2 with net resorption of sodium ions paragraph (3 steps)
  • SGLT (2nd last paragraph of page 345) (mcqs)
  • Fig 28.3
  • Transport maximum,tubular load and threshold
  • Transport max of glucose value
  • Diff btw transport maximum and renal threshold of glucose ( google)
  • Fig. 28.4 (seq)
  • Fig 28.5 28.6 28.8 28.9 28.10 28.11
  • Fig 28.12, 28.13
  • Glomerulotubular balance
  • Table 28.2 (read)
  • Table 28.3 (2023 proff SEQ)
  • Fig 28.18 28.19
  • Use of clearance method whole topic (vvvvimp)
  • Table 28.4
  • Osmolarity low and high
  • Renal mechanism for dilute urine
  • Fig 29.1
  • Fig 29.2
  • Obligatory urine volume
  • Facultative resorption ( the resorption of water that take place in late distal tubule and cortical tubule under the influence of ADH)
  • Sea water cause dehydration
  • Excreting conc.urine requirements
  • Countercurrent mechanism complete + steps involved (vvvv imp with fig 29.4)
  • Fig 29.5
  • Urea contributes to hyperosmosis (imp)
  • Fig 29.6
  • Countercurrent exchanger by vasa recta
  • Fig 29.7
  • Free water and osmolar clearance
  • Disorders of urinary conc abilities all (vvvvvvv imp) diabetes insipidus
  • Fig 29.9 (seq)
  • Table 29.2
  • Table 29.3
  • Disorders of thirst and water intake (blue box)
  • Role of angiotensin and aldosterone in controlling ecf osmolarity
  • Table 30.1
  • Fig 30.2
  • Fig 30.7
  • Fig 30.10
  • Control of calcium excretion (fig 30.11)
  • Table 30.2
  • Table 30.3
  • Table 30.4
  • Pg 393-399 (read flowcharts from medical gateway)
  • Page 398 (renal escape in 2nd blue box)
  • Conditions causing increase in ECF( pg 400)
  • Acid base regulation ( don’t need to do it again… biochemistry wla hi yahan revise kr lein)
  • Isohydric principal in blue box (pg 408)
  • How Kidneys regulate ecf H+ concentration? (3 points on pg 410)
  • Secretion of H+ and absorption of HCO3
  • Fig 31.5
  • Table 31.2
  • Clinical causes of acid base disorders (last blue box) read it thoroughly and shortlist it
  • Treatment of acidosis and alkalosis
  • Anion gap(vvvvimp) complete
  • Table 32.1 (vvvvimp)
  • Kidney disease (acute and chronic)
  • Acute kidney injury with table 32.2
  • Glomerulonephritis
  • Chronic kidney disease with Table 32.4
  • Fig 32.2
  • Glomerulonephritis by chronic kidney disease
  • Injury to renal interstitium
  • Nephrotic syndrome
  • Effects of renal failure (Uremia)
  • Fanconi syndrome, barter syndrome, gitelman syndrome, liddle syndrome (page 433)

Leave a Comment